Unit Synopsis
On completion of this unit students will be able to appraise the health consequences of defects in molecular and cellular mechanisms that lead to the development of diseases such as degenerative diseases of the nervous, respiratory and cardiovascular systems, cancer, diabetes and chronic inflammation. Tissue responses to stress and regenerative healing mechanisms will be discussed. Distance education (FLEX) students will be required to have access to a computer to make frequent use of internet resources.
Details
| Level | Undergraduate |
|---|---|
| Unit Level | 3 |
| Credit Points | 6 |
| Student Contribution Band | SCA Band 2 |
| Fraction of Full-Time Student Load | 0.125 |
| Pre-requisites or Co-requisites |
Prerequisite: BIOL12106 Molecular Biology or BMSC12010 Clinical Biochemistry or BMED19003 Clinical Biochemistry Important note: Students enrolled in a subsequent unit who failed their pre-requisite unit, should drop the subsequent unit before the census date or within 10 working days of Fail grade notification. Students who do not drop the unit in this timeframe cannot later drop the unit without academic and financial liability. See details in the Assessment Policy and Procedure (Higher Education Coursework). |
| Class Timetable | View Unit Timetable |
| Residential School | No Residential School |
Unit Availabilities from Term 1 - 2026
Attendance Requirements
All on-campus students are expected to attend scheduled classes - in some units, these classes are identified as a mandatory (pass/fail) component and attendance is compulsory. International students, on a student visa, must maintain a full time study load and meet both attendance and academic progress requirements in each study period (satisfactory attendance for International students is defined as maintaining at least an 80% attendance record).
Recommended Student Time Commitment
Each 6-credit Undergraduate unit at CQUniversity requires an overall time commitment of an average of 12.5 hours of study per week, making a total of 150 hours for the unit.
Assessment Tasks
| Assessment Task | Weighting |
|---|---|
| 1. Examination | 40% |
| 2. Written Assessment | 60% |
This is a graded unit: your overall grade will be calculated from the marks or grades for each assessment task, based on the relative weightings shown in the table above. You must obtain an overall mark for the unit of at least 50%, or an overall grade of ‘pass’ in order to pass the unit. If any ‘pass/fail’ tasks are shown in the table above they must also be completed successfully (‘pass’ grade). You must also meet any minimum mark requirements specified for a particular assessment task, as detailed in the ‘assessment task’ section (note that in some instances, the minimum mark for a task may be greater than 50%).
Past Exams
All University policies are available on the Policy web site, however you may wish to directly view the following policies below.
This list is not an exhaustive list of all University policies. The full list of policies are available on the Policy web site .
Term 1 - 2015 : The overall satisfaction for students in the last offering of this course was 100.00% (`Agree` and `Strongly Agree` responses), based on a 42.86% response rate.
Feedback, Recommendations and Responses
Every unit is reviewed for enhancement each year. At the most recent review, the following staff and student feedback items were identified and recommendations were made.
Source: Course Evaluation
'Assessment was a good learning tool'
Continue with similar assessment items.
In Progress
Source: Course Evaluation
One student suggested lecture slides could be better structured.
Will check on slides with a view to structuring better where required.
In Progress
On successful completion of this unit, you will be able to:
- Evaluate current theories of tumourgenicity including concepts of oncogenes and tumour suppressor genes.
- Appraise current understanding of the cellular and molecular mechanisms underlying muscular dystrophies.
- Review cellular and molecular mechanisms underlying degenerative diseases of the respiratory, cardiovascular and nervous systems.
- Present examples of channelopathies that result in disease.
- Appraise anomalies of enzyme activity responsible for pathophysiological outcomes.
- Appraise the role of receptor function in disease processes.
- Explain hypersensitivity disorders at the molecular level.
- Discuss effect of anomalies of synaptic transmission.
- Establish the effects of mitochondrial dysfunction and the relationship to oxidative stress injury.
- Critically evaluate the current scientific literature and present a detailed review of molecular causes of one of the above diseases.
| Assessment Tasks | Learning Outcomes | |||||||||
|---|---|---|---|---|---|---|---|---|---|---|
| 1 | 2 | 3 | 4 | 5 | 6 | 7 | 8 | 9 | 10 | |
| 1 - Examination | • | • | • | • | • | • | • | • | • | |
| 2 - Written Assessment | • | |||||||||
| Graduate Attributes | Learning Outcomes | |||||||||
|---|---|---|---|---|---|---|---|---|---|---|
| 1 | 2 | 3 | 4 | 5 | 6 | 7 | 8 | 9 | 10 | |
| 1 - Communication | • | • | • | • | • | • | • | • | • | |
| 3 - Critical Thinking | • | • | • | • | • | |||||
| 4 - Information Literacy | • | • | • | • | • | • | • | • | • | • |
| Assessment Tasks | Graduate Attributes | ||||||||||
|---|---|---|---|---|---|---|---|---|---|---|---|
| 1 | 2 | 3 | 4 | 5 | 6 | 7 | 8 | 9 | 10 | 10 | |